Role of glucocorticoids and Toxoplasma gondii infection on murine intestinal epithelial cells

Keyphrases

• Intestinal Epithelial Cells 100%
• Toxoplasma Gondii Infection 100%
• Glucocorticoid-induced Leucine Zipper 100%
• Corticosterone 66%
• MODE-K Cells 50%
• Toxoplasma Gondii (T. gondii) 33%
• Epithelial Cells 33%
• Chemokines 33%
• T. Gondii Infection 16%
• Toll-like 16%
• Stress Hormones 16%
• Infected Mice 16%
• Pre-exposure 16%
• Protein Drug 16%
• Cell Response 16%
• Physiological Stress 16%
• CXCL10 16%
• Chemokine (C-C motif) Ligand 2 (CCL2) 16%
• Receptor Expression 16%
• Inflammatory Chemokines 16%
• CXCL8 16%
• Infected Cells 16%
• Tissue Inflammation 16%
• IP-10 16%
• MIP-2 16%
• Endogenous Glucocorticoids 16%
• Decreased Expression 16%
• Monocyte Chemoattractant protein-1 (MCP-1) 16%
• Anti-inflammatory Molecules 16%
• Protozoan Parasites 16%
• Parasite Replication 16%
• Immunoblotting 16%
• RNA Therapy 16%
• Infection Exposure 16%

Immunology and Microbiology

• Glucocorticoid 100%
• Toxoplasmosis 100%
• Chemokine 30%
• Polymerase Chain Reaction 10%
• Toll-Like Receptor 10%
• Toxoplasma gondii 10%
• Cytokine 10%
• Mouse 10%
• Upregulation 10%
• Western Blot 10%
• Physiological Stress 10%
• CXCL10 10%
• CCL2 10%
• Protozoa 10%