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Evidence for the receipt of DNA damage stimuli by PML nuclear domains
A. Varadaraj
, C. L. Dovey
, L. Laredj
, B. Ferguson
, C. E. Alexander
, N. Lubben
, A. H. Wylie
, T. Rich
Research output
:
Contribution to journal
›
Article
›
peer-review
33
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Scopus citations
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Keyphrases
DNA Damage
100%
Nuclear Domains
100%
Promyelocytic Leukemia
92%
Ionizing Radiation
69%
Fibroblasts
23%
DNA Damage Response
15%
Signaling Pathway
7%
Swift
7%
Cell Cycle Arrest
7%
Growth Potential
7%
Cell Cycle Progression
7%
Apoptosis
7%
HCT116 Cells
7%
Irradiation
7%
DNA Damage Signaling
7%
Late Loss
7%
Cargo Protein
7%
Co-regulation
7%
Null Cell
7%
HCT116 Colon Cancer Cells
7%
Ionizing Radiation Induced Foci
7%
Cell Cycle Distribution
7%
Cargo
7%
Cell State
7%
Cell Failure
7%
Checkpoint Kinase 2 (CHEK2)
7%
Senescent Cells
7%
Biochemistry, Genetics and Molecular Biology
DNA Damage
100%
Polymorphonuclear Cell
100%
Fibroblast
60%
DNA Damage Response
40%
Signal Transduction
20%
Cancer Cell
20%
Cell Cycle Progression
20%
Cell Cycle
20%
Cell Cycle Checkpoint
20%
Promyelocytic Leukemia Protein
20%
Cell Cycle Arrest
20%
Colon
20%
Programmed Cell Death
20%
Neuroscience
DNA Damage
100%
Polymorphonuclear Cell
100%
Fibroblast
60%
DNA Damage Response
40%
Cell Signaling
20%
Cell Cycle
20%
Promyelocytic Leukemia Protein
20%
Cell Cycle Progression
20%
Cell Cycle Arrest
20%
Nucleus
20%
Programmed Cell Death
20%
Cell Cycle Checkpoint
20%